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Dysarthria is commonly caused by neurological conditions such
as Parkinsons disease, Cerebral Palsy, Lou Gehrigs
disease, or late stages of multiple sclerosis. It may also
occur after a cerebral vascular
accident, head trauma,
tumor, or infection. Existing at birth or occurring over time,
dysarthria is considered a disorder of movement. Symptoms
occur due to paralysis, weakness, or incoordination of speech
| Unilateral damage may be noticed in facial features
that droop on the side of the face opposite to the side
of the brain where the lesion occured. |
musculature and can be noticed in any of the components required for fluent speech.
These include respiration (breathing), phonation (sound produced
from
the vibration of the vocal folds), resonance (the inflection
or tone of voice), articulation (movement of oral structures)
and prosody (quality and intensity of voice).
Dysarthria
resulting from a stroke is generally broken into two categories
based on site of lesion. Damage to upper motor neurons, UMN,
those running from the brain to the spine, results in spastic
dysarthria, while damage to the lower motor neurons, LMN,
those running from the spine to the muscle, results in flaccid
dysarthria.
Meaning
"excessive tone," spastic dysarthria damage can
be unilateral or bilateral.
Unilateral
damage may be noticed in facial features that droop on the
side of the face opposite the site of the brain lesion. The
tongue often moves slowly and has difficulty meeting the weak
side of the mouth (the side opposite the lesion). The palate,
or portion of the oral cavity that forms the roof of the mouth,
is usually affected only minimally. The larynx tends to be
intact so swallow function is often preserved. Prognosis for
intelligible speech can be good if therapy is initiated within
a brief period of time after onset.
A
more devastating side of spastic dysarthria occurs with bilateral
UMN damage. Also known as pseudobulbar palsy, bilateral UMN
damage severely affects both range and rate of articulation
movement. The back and front of the tongue tend to be disproportionately
affected, causing different classes of sounds to be distorted.
Voice quality may appear "harsh," sounding almost
strangled. Pitch of voice is commonly low with little variation
in loudness. It is not uncommon for chewing and swallowing
problems to arise. Movement of the palate can be severely
reduced, causing excessively nasal speech.
Caused
by any disease that affects a part of the motor unit, LMN
damage is known as flaccid dysarthria. Symptoms may be seen
in reflexive, automatic, or voluntary movement and most commonly
arise from a brainstem stroke or condition known as myasthenia
gravis. Reflexes become reduced, which in turn shortens or
causes atrophy to the muscle over time. If atrophy occurs,
muscles may become flabby and have difficulty lifting, reaching,
or moving forward. Tiny tremors may also occur, disrupting
the fluency of movements, especially in the tongue. Speech
production will range in its precision, but sounds requiring
the tongue tip (/p/, /t/, and /k/) or intraoral pressure (/s/,
/f/) are more susceptible to error.
Damage
to the vocal fold will also affect voice production. If one
of the folds is paralyzed, quality of speech depends on the
position of the fold when it became paralyzed. More common
than a single fold being paralyzed, both may be unable to
move. If this is the case, a patient may have trouble changing
pitch or loudness, and voice will often be breathy. Other
characteristics of bilateral vocal fold paralysis are audible
inhalation and speaking in short phrases.
Another
area of concern for patients with lower motor neuron damage
is that of dysphagia,
or swallowing. Injuries to the brainstem are commonly related
to ability to protect the airway. Because brainstem strokes
commonly cause flaccid dysarthria, many patients with lower
motor neuron damage develop dysphagia.
If
both upper and lower motor neurons are involved, a patient
is diagnosed with Amyotrophic Lateral Sclerosis (ALS), or
Lou Gehrigs disease, usually occuring in the fifth decade
of life, ALS has various symptoms depending on the type and
extent of neurons affected.
Speech
therapists at the Center for Speech, Language, and Occupational
Therapy, Inc. currently provide several options for alternative
communication and feeding. Because each patient is unique,
professionals design a therapy program to best suit the individuals
immediate needs. If deficits exist in both speech and swallowing,
the swallowing issues will be addressed first to establish
proper nutrition. If an individual is strong, both areas can
be addressed at the same time. It is important to remember
that differences in symptoms and therapy potential will occur
depending on site and severity of lesion.
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